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Julia B Jernberg

  • Associate Clinical Professor, Medicine - (Clinical Series Track)
  • Associate Professor, Public Health
  • Clinical Associate Professor, (Clinical Series Track)
Contact
  • (520) 626-9660
  • AHSC, Rm. 2301
  • TUCSON, AZ 85724-5099
  • jbj1@arizona.edu
  • Bio
  • Interests
  • Courses
  • Scholarly Contributions

Biography

Internal Medicine physician with Geriatrics and Integrative Medicine specialization. Interested in climate, the environment, One Health, education, program development. 

Degrees

  • M.B.A.
    • Eller College Univ of Arizona, Tucson, Arizona
  • M.D. Medicine
    • University of Illinois, Chicago, Illinois, United States

Work Experience

  • University of Arizona College of Medicine- Banner Tucson (2019 - Ongoing)
  • Trader Joes (2018 - Ongoing)
  • SAVAHCS (So. AZ VA) (2016 - 2018)
  • Iora Health (2015 - 2016)
  • Casa de la Luz Hospice (2013 - 2015)
  • Univ of Arizona Health Network (2012 - 2013)
  • UA Center for Integrative Medicine (2009 - 2011)
  • SAVAHCS via Medical Doctor Assoc (2009)
  • MedMinds, PLLC and self (2003 - Ongoing)
  • UA Center for Integrative Medicine (2003 - 2015)
  • Univ of Wisconsin-Madison (1998 - 2013)
  • Group Health Cooperative (1997 - 1998)

Awards

  • Specialty Advisor Adward
    • UACOM-T, Spring 2024
  • Virgninia and Vernon Furrows Award for Excellence in Innovation in Teaching
    • Furrows via UACOM-T, Spring 2023

Licensure & Certification

  • ABIM Specialty: Geriatrics (2020)
  • American Board of Integrative Medicine, ABOIM (2017)
  • American Board of Internal Medicine, ABIM (2014)

Related Links

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Interests

Research

ClimateEnvironmentOne Health

Teaching

Interested in problem solving and creative thinking, within the context of critical analysesClimate Environment Public Health/One Health

Courses

2024-25 Courses

  • Ambulatory Medicine
    MEDI 850O (Spring 2025)
  • Climate, Envr. and Health
    MED 850A (Spring 2025)
  • Independent Study
    MED 899 (Spring 2025)
  • Thesis
    CMM 910 (Spring 2025)
  • Ambulatory Medicine
    MEDI 850O (Fall 2024)
  • Independent Study
    MED 899 (Fall 2024)

2023-24 Courses

  • Ambulatory Medicine
    MEDI 850O (Spring 2024)
  • Climate, Envr. and Health
    MED 850A (Spring 2024)
  • Independent Study
    MED 899 (Spring 2024)
  • Ambulatory Medicine
    MEDI 850O (Fall 2023)
  • Independent Study
    MED 899 (Fall 2023)

2022-23 Courses

  • Ambulatory Medicine
    MEDI 850O (Spring 2023)
  • Independent Study
    MED 899 (Spring 2023)
  • Ambulatory Medicine
    MEDI 850O (Fall 2022)
  • Clinical Reasoning: An Intro
    MED 101 (Fall 2022)

2021-22 Courses

  • Clinical Reasoning: An Intro
    MED 101 (Spring 2022)
  • Ambulatory Medicine
    MEDI 850O (Fall 2021)
  • Ambulatory Medicine Clinical
    MEDI 850O2 (Fall 2021)
  • Independent Study
    MED 899 (Fall 2021)

2020-21 Courses

  • Ambulatory Medicine
    MEDI 850O (Spring 2021)

Related Links

UA Course Catalog

Scholarly Contributions

Journals/Publications

  • Klotz, S. A., Jernberg, J. B., & Robbins, R. A. (2023). Turn Healthcare Workers Loose with Outpatient Telemedicine-Let Them Decide Its Fate; No Top-Down Decisions on What It Can and Cannot Do. The American journal of medicine, 136(10), 955-957.
  • Najjar, S. M., Accili, D., Philippe, N., Jernberg, J., Margolis, R., & Taylor, S. I. (1993). pp120/ecto-ATPase, an endogenous substrate of the insulin receptor tyrosine kinase, is expressed as two variably spliced isoforms. The Journal of biological chemistry, 268(2), 1201-6.
    More info
    The insulin receptor possesses tyrosine kinase activity which is thought to mediate the biological effects of insulin upon target cells. pp120 is a liver-specific glycoprotein of apparent molecular size of 120 kDa that is phosphorylated on tyrosine residues by the receptors for insulin, insulin-like growth factor-I, and epidermal growth factor. Previously, we demonstrated that pp120 is identical to a liver-specific ecto-ATPase. In the present study, we have cloned the rat gene encoding pp120/ecto-ATPase. The gene is contained within approximately 15 kilobases of genomic DNA, and consists of nine exons interrupted by eight introns. Using the reverse transcriptase/polymerase chain reaction, we isolated cDNA clones complementary to rat liver mRNA encoding pp120/ecto-ATPase. Sequence analysis indicated the presence of two populations of cDNA's that differ by the presence or absence of a 53-base pair (bp) fragment encoding the juxta-membrane region of the cytoplasmic domain. By cloning the corresponding region of the ecto-ATPase gene, we demonstrated that the 53-bp represents exon 7 of the gene. This 53-bp exon undergoes alternative splicing, thereby giving rise to two mRNA variants. Deletion of this 53-bp cassette exon introduces a frameshift, and results in a premature chain termination codon that truncates the cytoplasmic domain. The truncated cytoplasmic domain contains 10 rather than 71 amino acid residues. Because the short isoform of ecto-ATPase lacks the putative sites for tyrosine- and serine-specific phosphorylation, this alternative splicing may have a major effect upon the physiological function of the enzyme.
  • Ambrosino, D. M., Siber, G. R., Chilmonczyk, B. A., Jernberg, J. B., & Finberg, R. W. (1987). An immunodeficiency characterized by impaired antibody responses to polysaccharides. The New England journal of medicine, 316(13), 790-3.

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