Farshad Fani Marvasti
- Associate Professor, Academic Affairs (Clinical Scholar Track)
- Associate Professor, Family / Community and Preventive Medicine - (Clinical Scholar Track)
- Director, Public Health / Prevention / Population Health Theme
- Co-Director, Capstones
- Physician, Doctoring Program
- Facilitator, Longitudinal Case-Based Instruction
- Associate Professor, Nutritional Sciences and Wellness
No activities entered.
No activities entered.
CapstoneMEDP 817A-T2 (Spring 2024)
CapstoneMEDP 817A-T1 (Fall 2023)
Capstone MS2MEDP 817B-T1 (Fall 2023)
CapstoneMEDP 817A-T2 (Spring 2023)
CapstoneMEDP 817A-T1 (Fall 2022)
Capstone MS2MEDP 817B-T1 (Fall 2022)
CapstoneMEDP 817A-T2 (Spring 2022)
Capstone MS2MEDP 817B-T2 (Spring 2022)
CapstoneMEDP 817A-T1 (Fall 2021)
Capstone MS2MEDP 817B-T1 (Fall 2021)
- Fani Marvasti, F. (2018). The Role of Complementary and Alternative Medicine in Integrative Pre-ventive Medicine. In Textbook of Integrative Preventive Medicine. Oxford University Press.
- Fani Marvasti, F. (2018). The Role of Family and Community in Integrative Preventive Medicine. In Textbook of Integrative Preventive Medicine. Oxford University Press.
- Stafford, R. S., & Marvasti, F. F. (2012). From sick care to health care--reengineering prevention into the U.S. system.. The New England journal of medicine, 367(10), 889-91. doi:10.1056/nejmp1206230More infoFlexner's acute care model remains securely embedded in the U.S. health care system. But given our chronic-disease epidemics, unsustainable costs, poor outcomes, frequent medical errors, and worsening health disparities, we must replace it with a prevention model.
- Dewell, A., Marvasti, F. F., Harris, W. S., Gardner, C. D., & Tsao, P. S. (2011). Low- and high-dose plant and marine (n-3) fatty acids do not affect plasma inflammatory markers in adults with metabolic syndrome.. The Journal of nutrition, 141(12), 2166-71. doi:10.3945/jn.111.142240More infoChronic inflammation is considered to play a role in the development of cardiovascular disease. Various (n-3) fatty acids (FA) have been reported to have antiinflammatory effects, but there is a lack of consensus in this area, particularly in regard to optimal source(s) and dose(s). This study aimed to determine the effects of high and low doses of (n-3) FA from plant and marine sources on plasma inflammatory marker concentrations. One-hundred adults with metabolic syndrome were randomly assigned to a low or high dose of plant- (2.2 or 6.6 g/d α-linolenic acid) or marine- (1.2 or 3.6 g/d EPA and DHA) derived (n-3) FA or placebo for 8 wk, using a parallel arm design (n = 20/arm). Fasting blood samples collected at 0, 4, and 8 wk were analyzed for concentrations of monocyte chemotactic protein-1 (MCP-1), IL-6, and soluble intercellular adhesion molecule-1 (sICAM-1) and for cardiovascular risk factors. Baseline concentrations across all 5 groups combined were (mean ± SD) 103 ± 32 ng/L for MCP-1, 1.06 ± 0.56 ng/L for IL-6, and 0.197 ± 0.041 ng/L for sICAM-1. There were no significant differences in 8-wk changes in plasma inflammatory marker concentrations among the 5 groups. Plasma TG and blood pressure decreased significantly more and the LDL cholesterol concentration increased more in the high-dose fish oil group compared to the 8-wk changes in some of the other 4 groups (P ≤ 0.04). In conclusion, no beneficial effects were detected for any of the 3 inflammatory markers investigated in response to (n-3) FA in adults with metabolic syndrome regardless of dose or source.