Talal Moukabary

Interests

Teaching

Electrocardiogram interpretation, Cardiac anatomy, Clinical Cardiology and Cardiac electrophysiology, Clinical Reasoning, Computer programming for simulation of cardiac arrhythmia

Research

Stem cell and iPS (induced pluripotent stem) cell therapies for heart failure and cardiac arrhythmia. Long Covid. Postural orthostatic tachycardia syndrome. Acoustoelectric imaging, Computer modeling of cardiac arrhythmia, software and hardware development of implanted and wearable cardiac devices.

Courses

Scholarly Contributions

Journals/Publications

• , ., Shah, D. P., Thaweethai, T., Karlson, E. W., Bonilla, H., Horne, B. D., Mullington, J., Wisnivesky, J., Hornig, M., Shinnick, D., Klein, J. D., Berdmann, N., Brosnahan, S., Lee-Iannotti, J., Metz, T., Maughan, C., Ofotokun, I., Reeder, H., Stiles, L., , Shaukat, A., et al. (2024). Sex differences in PASC. JAMA Network Open.
• Lancaster, J. J., Grijalva, A., Fink, J., Benson, D., Ref, J., Fox, K., Mangiola, M., Pandey, A. C., Zile, M. R., Mann, D. L., Perin, E. C., Singh, S., Moukabary, T., Koevary, J. W., & Goldman, S. (2024). Immune Modulation: New Treatment for Heart Failure. Immunome Research, 20(1), 257.
• Mostafizi, P., Lefkowitz, E., Ref, J., Daugherty, S., Grijalva, A., Cook, K. E., Chinyere, I., Lancaster, J., Koevary, J., Goldman, S., & Moukabary, T. (2024). Abstract 4114694: A Large Animal Model of Persistent Atrial Fibrillation. Circulation, 150(Suppl_1).
• Goldman, S., Koevary, J. W., Bradshaw, A., Zile, M., Pandey, A., Mangiola, M., Benson, D., Borgstrom, M., Moukabary, T., Pierce, M. L., Strom, J., McArthur, A., Acharya, T., Avery, R., Lancaster, L., Gorman, G., Fox, K., Whitman, S., Daugherty, S., , Ref, J., et al. (2023). Biologically derived epicardial patch induces macrophage mediated pathophysiologic repair in chronically infarcted swine hearts. Communications Biology.
• Chinyere, I. R., Hutchinson, M. D., Moukabary, T., Goldman, S., Lancaster, J. J., Juneman, E., & Koevary, J. W. (2021). Modulating the Infarcted Ventricle’s Refractoriness with an Epicardial Biomaterial. Journal of Investigative Medicine, 69(2), 364-370. doi:10.1136/jim-2020-001486
• Chinyere, I. R., Moukabary, T., Goldman, S., & Juneman, E. (2021). Electrical and mechanical alternans during ventricular tachycardia with moderate chronic heart failure. Journal of electrocardiology, 51(1), 33-37.
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  A chronic heart failure (CHF) rat underwent epicardial programmed electrical stimulation (PES). Ventricular tachycardia (VT) developed during PES. Mechanical alternans was noted despite fixed tachycardia cycle length. Anti-tachycardia pacing attempts initiated a second VT that generated pulse intermittently and then degenerated into pulseless VT with electrical alternans.To our knowledge electrical and mechanical alternans have not been recorded in animal models of CHF during VT. The distinct events of mechanical alternans and electrical alternans may be indicative of progressively worsened calcium handling in the compromised cardiomyocytes.Although ion channel differences between rodents and humans exist, this work attempts to demonstrate this rat model's usefulness in understanding cardiac electrophysiology in CHF.
• Chinyere, I. R., Moukabary, T., Hutchinson, M. D., Lancaster, J. J., Juneman, E., & Goldman, S. (2021). Progression of infarct-mediated arrhythmogenesis in a rodent model of heart failure. American Journal of Physiology-heart and Circulatory Physiology. doi:10.1152/ajpheart.00639.2020
• Chinyere, I. R., Moukabary, T., Hutchinson, M. D., Lancaster, J. J., Juneman, E., & Goldman, S. (2021). Progression of infarct-mediated arrhythmogenesis in a rodent model of heart failure. American journal of physiology. Heart and circulatory physiology, 320(1), H108-H116.
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  Heart failure (HF) post-myocardial infarction (MI) presents with increased vulnerability to monomorphic ventricular tachycardia (mmVT). To appropriately evaluate new therapies for infarct-mediated reentrant arrhythmia in the preclinical setting, chronologic characterization of the preclinical animal model pathophysiology is critical. This study aimed to evaluate the rigor and reproducibility of mmVT incidence in a rodent model of HF. We hypothesize a progressive increase in the incidence of mmVT as the duration of HF increases. Adult male Sprague-Dawley rats underwent permanent left coronary artery ligation or SHAM surgery and were maintained for either 6 or 10 wk. At end point, SHAM and HF rats underwent echocardiographic and invasive hemodynamic evaluation. Finally, rats underwent electrophysiologic (EP) assessment to assess susceptibility to mmVT and define ventricular effective refractory period (ERP). In 6-wk HF rats ( = 20), left ventricular (LV) ejection fraction (EF) decreased ( < 0.05) and LV end-diastolic pressure (EDP) increased ( < 0.05) compared with SHAM ( = 10). Ten-week HF ( = 12) revealed maintenance of LVEF and LVEDP ( > 0.05), ( > 0.05). Electrophysiology studies revealed an increase in incidence of mmVT between SHAM and 6-wk HF ( = 0.0016) and ERP prolongation ( = 0.0186). The incidence of mmVT and ventricular ERP did not differ between 6- and 10-wk HF ( = 1.0000), ( = 0.9831). Findings from this rodent model of HF suggest that once the ischemia-mediated infarct stabilizes, proarrhythmic deterioration ceases. Within the 6- and 10-wk period post-MI, no echocardiographic, invasive hemodynamic, or electrophysiologic changes were observed, suggesting stable HF. This is the necessary context for the evaluation of experimental therapies in rodent HF. Rodent model of ischemic cardiomyopathy exhibits a plateau of inducible monomorphic ventricular tachycardia incidence between 6 and 10 wk postinfarction.
• Chinyere, I. R., Moukabary, T., Hutchinson, M., Koevary, J. W., Paulino, L., Juneman, E., Goldman, S., & Lancaster, J. (2021). Localized Ventricular effective Refractory period Prolongation with an Epicardial Biomaterial. Journal of Investigative Medicine, 69(2), 364-370.
• Chinyere, I. R., Hutchinson, M., Moukabary, T., Koevary, J. W., Juneman, E., Goldman, S., & Lancaster, J. J. (2020). Modulating the Infarcted Ventricle's Refractoriness with an Epicardial Biomaterial. Journal of investigative medicine : the official publication of the American Federation for Clinical Research.
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  Patients diagnosed with heart failure with reduced ejection fraction (HFrEF) are at increased risk of monomorphic ventricular tachycardia (VT) and ventricular fibrillation. The presence of myocardial fibrosis provides both anatomical and functional barriers that promote arrhythmias in these patients. Propagation of VT in a reentrant circuit depends on the presence of excitable myocardium and the refractoriness of the circuit. We hypothesize that myocardial refractoriness can be modulated surgically in a model of HFrEF, leading to decreased susceptibility to VT.Male Sprague-Dawley rats were infarcted via permanent left coronary artery ligation. At 3 weeks post-infarction, engineered grafts composed of human dermal fibroblasts cultured into a polyglactin-910 biomaterial were implanted onto the epicardium to cover the area of infarction. Three weeks post-graft treatment, all rats underwent a terminal electrophysiologic study to compare monophasic action potential electroanatomic maps and susceptibility to inducible monomorphic VT.HFrEF rats (n=29) demonstrated a longer (p=0.0191) ventricular effective refractory period (ERP) and a greater (p=0.0394) VT inducibility compared with sham (n=7). HFrEF rats treated with the graft (n=12) exhibited no change in capture threshold (p=0.3220), but had a longer ventricular ERP (p=0.0029) compared with HFrEF. No statistically significant change in VT incidence was found between HFrEF rats treated with the graft and untreated HFrEF rats (p=0.0834).Surgical deployment of a fibroblast-containing biomaterial in a rodent ischemic cardiomyopathy model prolonged ventricular ERP as measured by programmed electrical stimulation. This hypothesis-generating study warrants additional studies to further characterize the antiarrhythmic or proarrhythmic effects of this novel surgical therapy.
• Chinyere, I. R., Hutchinson, M. D., Moukabary, T., Lancaster, J. J., Juneman, E., & Goldman, S. (2019). Monophasic action potential amplitude for substrate mapping. American Journal of Physiology-heart and Circulatory Physiology. doi:10.1152/ajpheart.00225.2019
• Chinyere, I. R., Hutchinson, M., Moukabary, T., Lancaster, J., Goldman, S., & Juneman, E. (2019). Monophasic action potential amplitude for substrate mapping. American journal of physiology. Heart and circulatory physiology, 317(4), H667-H673.
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  Although radiofrequency ablation has revolutionized the management of tachyarrhythmias, the rate of arrhythmia recurrence is a large drawback. Successful substrate identification is paramount to abolishing arrhythmia, and bipolar voltage electrogram's narrow field of view can be further reduced for increased sensitivity. In this report, we perform cardiac mapping with monophasic action potential (MAP) amplitude. We hypothesize that MAP amplitude (MAPA) will provide more accurate infarct sizes than other mapping modalities via increased sensitivity to distinguish healthy myocardium from scar tissue. Using the left coronary artery ligation Sprague-Dawley rat model of ischemic heart failure, we investigate the accuracy of in vivo ventricular epicardial maps derived from MAPA, MAP duration to 90% repolarization (MAPD), unipolar voltage amplitude (UVA), and bipolar voltage amplitude (BVA) compared with gold standard histopathological measurement of infarct size. Numerical analysis reveals discrimination of healthy myocardium versus scar tissue using MAPD ( = 0.0158) and UVA ( < 0.001, = 21). MAPA and BVA decreased between healthy and border tissue ( = 0.0218 and 0.0015, respectively) and border and scar tissue ( = 0.0037 and 0.0094, respectively). Contrary to our hypothesis, BVA mapping performed most accurately regarding quantifying infarct size. MAPA mapping may have high spatial resolution for myocardial tissue characterization but was quantitatively less accurate than other mapping methods at determining infarct size. BVA mapping's superior utility has been reinforced, supporting its use in translational research and clinical electrophysiology laboratories. MAPA may hold potential value for precisely distinguishing healthy myocardium, border zone, and scar tissue in diseases of disseminated fibrosis such as atrial fibrillation. Monophasic action potential mapping in a clinically relevant model of heart failure with potential implications for atrial fibrillation management.
• Lancaster, J. J., Sanchez, P., Repetti, G. G., Juneman, E., Pandey, A. C., Chinyere, I. R., Moukabary, T., LaHood, N., Daugherty, S. L., & Goldman, S. (2019). Human Induced Pluripotent Stem Cell-Derived Cardiomyocyte Patch in Rats With Heart Failure. The Annals of thoracic surgery, 108(4), 1169-1177.
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  To treat chronic heart failure (CHF), we developed a robust, easy to handle bioabsorbable tissue-engineered patch embedded with human neonatal fibroblasts and human induced pluripotent stem cell-derived cardiomyocytes (hiPSC-CMs). This patch was implanted on the epicardial surface of the heart covering the previously infarcted tissue.
• Pandey, A. C., Moukabary, T., Goldman, S., Daugherty, S., Lahood, N., Chinyere, I. R., Juneman, E., Repetti, G. G., Sanchez, P., & Lancaster, J. J. (2019). Human Induced Pluripotent Stem Cell–Derived Cardiomyocyte Patch in Rats With Heart Failure. The Annals of Thoracic Surgery. doi:10.1016/j.athoracsur.2019.03.099
• Moukabary, T., Juneman, E., Goldman, S., Pierce, M. K., Lancaster, J. J., Chinyere, I., Witte, R. S., & Weigand, K. (2017). In vivo Electrophysiological Study of Induced Ventricular Tachycardia in Intact Rat Model of Chronic Ischemic Heart Failure. IEEE Transactions on Biomedical Engineering. doi:10.1109/tbme.2016.2605578
• Weigand, K., Witte, R., Moukabary, T., Chinyere, I., Lancaster, J., Pierce, M. K., Goldman, S., & Juneman, E. (2017). In vivo Electrophysiological Study of Induced Ventricular Tachycardia in Intact Rat Model of Chronic Ischemic Heart Failure. IEEE transactions on bio-medical engineering, 64(6), 1393-1399.
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  The objective of this study was to define the clinical relevance of in vivo electrophysiologic (EP) studies in a rat model of chronic ischemic heart failure (CHF).
• Moukabary, T., & Gonzalez, M. D. (2015). Management of atrial fibrillation. The Medical clinics of North America, 99(4), 781-94.
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  Atrial fibrillation is a very common clinical problem with a high prevalence that is expected to rise over time because of increasing risk factors (eg, age, obesity, hypertension). This high prevalence is also associated with high cost, because atrial fibrillation represents about 1% of overall health care spending. The management of atrial fibrillation involves multiple facets: (1) management of underlying disease if present and the management of atrial fibrillation risk factors, (2) prevention of thromboembolism, (3) control of the ventricular rate during atrial fibrillation, and (4) restoration and maintenance of normal sinus rhythm.
• Sriram, C. S., Banchs, J. E., Moukabary, T., Moradkhan, R., & Gonzalez, M. D. (2015). Detection of left atrial thrombus by intracardiac echocardiography in patients undergoing ablation of atrial fibrillation. Journal of interventional cardiac electrophysiology : an international journal of arrhythmias and pacing, 43(3), 227-36.
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  The role of intracardiac echocardiography (ICE) to detect thrombus within left atrium (LA) before atrial fibrillation (AF) ablation despite a recent transesophageal echocardiogram (TEE) is not well defined. We examined the prevalence of LA/left atrial appendage (LAA) thrombus using ICE immediately prior to AF ablation in patients in whom anticoagulation was not withheld.
• Márquez, M. F., Moukabary, T., & Gonzalez, M. D. (2014). Jesús Alanís and the first recording of the his bundle: the scientist and the man. Pacing and clinical electrophysiology : PACE, 37(12), 1724-7.
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  The anatomy and physiology of the specialized conduction system has intrigued investigators since the 19th century and is still not fully understood. Dr. Wilhelm His Jr. is well known because he discovered the A-V bundle, and Dr. Sunao Tawara is rightly credited with the discovery of the atrioventricular (AV) node, but who was the first to record the electrical activity of the His bundle? This paper reviews the historical background and scientific contributions made by Dr. Jesús Alanís in the middle of the 20th century working at the National Institute of Cardiology in Mexico City. Collaborating with outstanding investigators such as Arturo Rosenblueth, Dr. Alanís recorded for the first time the electrical activity of the His bundle in the isolate canine heart. That the recorded electrogram was indeed the His bundle and not the AV node was confirmed by detailed studies that set the basis for modern clinical electrophysiology. The life and research contributions of this extraordinary man are reviewed in the context of a unique group of investigators who made significant advances in cardiac electrophysiology.
• Márquez, M. F., Moukabary, T., & González, M. D. (2014).

  Jesús Alanís and the First Recording of the His Bundle: The Scientist and the Man

  . Pacing Clin Electrophysiol . 2014 Dec;37(12):1724-7. doi:10.1111/pace.12493
• Indik, J. H., Dallas, W. J., Gear, K., Tandri, H., Bluemke, D. A., Moukabary, T., & Marcus, F. I. (2012). Right ventricular volume analysis by angiography in right ventricular cardiomyopathy. The international journal of cardiovascular imaging, 28(5), 995-1001.
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  Imaging of the right ventricle (RV) for the diagnosis of arrhythmogenic right ventricular cardiomyopathy/dysplasia (ARVC/D) is commonly performed by echocardiography or magnetic resonance imaging (MRI). Angiography is an alternative modality, particularly when MRI cannot be performed. We hypothesized that RV volume and ejection fraction computed by angiography would correlate with these quantities as computed by MRI. RV volumes and ejection fraction were computed for subjects enrolled in the North American ARVC/D Registry, with both RV angiography and MRI studies. Angiography was performed in the 30° right anterior oblique (RAO) and 60° left anterior oblique (LAO) views. Angiographic volumes were computed by RAO view and two-view (RAO and LAO) formulae. 17 subjects were analyzed (11 men and 6 women), with 15 subjects classified as affected, and two as unaffected by modified Task Force criteria. The correlation coefficient of MRI to the two-view angiographic analysis was 0.72 (P = 0.003) for end-diastolic volume and 0.68 (P = 0.005) for ejection fraction. Angiographically derived volumes were larger than MRI derived volume (P = 0.009) and with the slope in a linear relationship equal to 0.8 for end diastolic volume, and 0.9 for RV ejection fraction (P < 0.001), computed by the two view formula. End-diastolic volumes and ejection fractions of the RV obtained by dual view angiography correlate with these quantities by MRI. RV end-diastolic volumes are larger by RV angiography in comparison with MRI.
• Moukabary, T., Faletra, F. F., Kronzon, I., Thomas, W., & Sorrell, V. L. (2012). Three-dimensional echocardiography in the electrophysiology laboratory. Echocardiography (Mount Kisco, N.Y.), 29(1), 117-22.
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  The use of three-dimensional echocardiography (3DE) during electrophysiology (EP) procedures is the end product of years of growth in two diverse cardiology subspecialties; namely, advanced cardiac imaging and the EP. During the past decade, progress in both fields has resulted in many important advances that have culminated in their union for a new area of growth and development. Imaging advances have provided the cardiovascular specialist with enhanced cardiac volume and function data, and more recently, 3DE capabilities with improved spatial and temporal resolution providing unprecedented spatial relationships. This latter development is valued by EP specialists in need of hitherto never required anatomic knowledge as they press forward with extraordinary expansion in their capabilities. It makes sense that by combining these two rapidly growing subspecialties, future capabilities in patient care may be achieved that would otherwise not be possible. This paper discusses the value of 3DE during EP procedures and offers the readers insight into this novel multispecialty hybrid arena. Using this model as a template, it is likely that the readers may identify other areas within their practices where periprocedural advanced imaging may afford significant dividends in patient outcomes.
• Moukabary, T., Marcus, F. I., Bluemke, D. A., Tandri, H., Gear, K., Dallas, W. J., & Indik, J. H. (2012). Right ventricular volume analysis by angiography in right ventricular cardiomyopathy. International Journal of Cardiovascular Imaging. doi:10.1007/s10554-011-9915-1
• Moukabary, T., Sorrell, V. L., Thomas, W. R., Kronzon, I., & Faletra, F. (2012). Three-Dimensional Echocardiography in the Electrophysiology Laboratory. Echocardiography-a Journal of Cardiovascular Ultrasound and Allied Techniques. doi:10.1111/j.1540-8175.2011.01613.x
• Moukabary, T., & Naccarelli, G. V. (2011). Prevention of Stroke in Patients With Atrial Fibrillation. Journal of atrial fibrillation, 4(4), 447.
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  The presence of atrial fibrillation (AF) increases the risk of stroke, especially in patients with risk factors as outlined by the CHADS2 and CHA2DS2-VASc scoring systems. Although warfarin can reduce stroke rates by over 65%, only 55% of patients, in the USA, who should be on warfarin for AF and stroke prevention are taking the drug due to the need of INR monitoring, difficulties in maintaining a therapeutic INR in the therapeutic range and dietary and drug interactions. Dabigatran, an oral direct thrombin inhibitor and rivaroxaban and apixaban, factor Xa inhibitors, have demonstrated efficacy in reducing stroke in large clinical trials. These novel anticoagulants will change the therapeutic landscape since patients will be able to prevent stroke with a lower risk of intracranial hemorrhage and without the need for INR monitoring and less drug-dietary interactions.
• Moukabary, T., & Naccarelli, G. V. (2011). Prevention of Stroke in Patients With Atrial Fibrillation.. Journal of Atrial Fibrillation. doi:10.447/jafib.447
• Moukabary, T., Thai, H., & Thal, S. (2011). Subclavian balloon venoplasty to facilitate lead implant in patient with subclavian venous obstruction. The Journal of invasive cardiology, 23(4), E83-5.
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  Implanting new leads for defibrillation or pacing leads could be problematic as a result of venous obstruction. Up to 50% of patients with pacemaker/implantable cardioverter defibrillator leads have an asymptomatic subclavian vein obstruction. Overcoming this obstruction could be very challenging. The current approaches of contralateral access, extraction and surgical intervention have significant drawbacks. This report presents an alternative approach that uses percutaneous subclavian balloon venoplasty successfully to regain venous access. We believe that this technique is safe and effective.
• Moukabary, T., Thal, S., & Thai, H. (2011). Subclavian balloon venoplasty to facilitate lead implant in patient with subclavian venous obstruction.. Journal of Invasive Cardiology.
• Moukabary, T., Hariri, S. A., Akoglu, A., & Nimmagadda, V. (2010). Cardiac simulation on multi-GPU platform. The Journal of Supercomputing,.
• Thal, S., Moukabary, T., Boyella, R., Shanmugasundaram, M., Pierce, M. K., Thai, H., & Goldman, S. (2010).

  The Relationship between Warfarin, Aspirin, and Clopidogrel Continuation in the Peri-procedural Period and the Incidence of Hematoma Formation after Device Implantation

  . Pacing Clin Electrophysiol . 2010 Apr;33(4):385-8.. doi:10.1111/j.1540-8159.2009.02674.x
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  Many patients requiring permanent pacemaker (PPM) or implantable cardiac defibrillator (ICD) placement are anticoagulated with warfarin, aspirin (ASA), and clopidogrel for a number of thromboembolic risk indications. The present review sought to evaluate the relationship between continuation of these medications in the peri-procedural period and the incidence of hematoma formation after implantation.We retrospectively reviewed consecutive patients undergoing PPM and ICD implantation at our hospital from January 2007-2009. All patients on warfarin, aspirin, and clopidogrel were maintained on these medications peri-operatively. We collected data on the use of warfarin at implantation, INR prior to device implantation, use of dual-antiplatelet therapy (DAPT), such as concomitant aspirin and clopidogrel and subsequent formation of hematoma in the peri-procedure period.PPM and ICD implantations were performed in 194 men and six women. The mean age was 73 years old. Fifty eight patients were taking warfarin with an average international normalized ratio of 1.9 +/- 0.6; 112 were on ASA, 23 on clopidogrel, and 20 of them on DAPT. Only five patients were on DAPT and warfarin combined at the time of device implantation. Hematomas formed in a total of seven patients (3.5%), five of whom were on DAPT consisting of ASA and clopidogrel (P < 0.0001) while only two of them were on warfarin (P = 0.67). Pocket revision for hematoma evacuation was needed in four patients (2%), three of whom were on DAPT and only one on warfarin.This study suggests that hematoma formation after PPM or ICD implantation is rare, even among those who are anticoagulated. There were more patients with hematoma on DAPT than warfarin therapy and half of these patients with this complication needed pocket revision for evacuation. (PACE 2010; 385-388).
• Thal, S., Moukabary, T., Boyella, R., Shanmugasundaram, M., Pierce, M. K., Thai, H., & Goldman, S. (2010). The relationship between warfarin, aspirin, and clopidogrel continuation in the peri-procedural period and the incidence of hematoma formation after device implantation. Pacing and clinical electrophysiology : PACE, 33(4), 385-8.
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  Many patients requiring permanent pacemaker (PPM) or implantable cardiac defibrillator (ICD) placement are anticoagulated with warfarin, aspirin (ASA), and clopidogrel for a number of thromboembolic risk indications. The present review sought to evaluate the relationship between continuation of these medications in the peri-procedural period and the incidence of hematoma formation after implantation.
• LaHood, N., & Moukabary, T. (2009). History of cardiopulmonary resuscitation. Cardiology journal, 16(5), 487-8.
• Moukabary, T., & Lahood, N. (2009). History of cardiopulmonary resuscitation.. Cardiology Journal.
• Moukabary, T. (2008). Understanding atrial fibrillation: a historical perspective. Cardiology journal, 15(4), 396-7.
• Moukabary, T. (2008). Understanding atrial fibrillation: a historical perspective.. Cardiology Journal.
• Moukabary, T., Hariri, S. A., Zhang, Y., & Jararweh, Y. (2008). Physics aware programming paradigm: approach and evaluation.. Proceedings of the 6th international workshop on Challenges of large applications in distributed environments.
• Moukabary, T. (2007). Willem Einthoven (1860-1927): Father of electrocardiography. Cardiology journal, 14(3), 316-7.

Proceedings Publications

• Bravo, F. W., Grijalva, A., Salciccioli, A., Benson, D., Pierce, M. K., Moukabary, T., Hortareas, J., Goldman, S., & Moukabary, T. (2025, January 16-18). Applying Raspberry Pi Technology to Induce Atrial Fibrillation in Swine. In Western Medical Research Conference.
• Mostafizi, P., Lefkowitz, E., Ref, J., Daugherty, S., Grijalva, A., Lancaster, J., Koewary, J. W., Goldman, S., & Moukabary, T. (2025, January 16-18). A Large Animal Model of Persistent Atrial Fibrillation. In Western Medical Research Conference.
• Moukabary, T., Hariri, S., Akoglu, A., & Szép, J. (2018). Two-Level Autonomous Optimizations Based on ML for Cardiac FEM Simulations. In 2018 proceedings article published by International Conference on Autonomic Computing..
• Hariri, S., Moukabary, T., Zhang, Y., & Jararweh, Y. (2008). Physics aware programming paradigm. In Proceedings of the 6th international workshop on Challenges of large applications in distributed environments.
• Moukabary, T., & Haines, D. (2007). Relationship between the potassium currents block and the occurrence of early after depolarizations in the setting of sodium current blockade. In 2007 Computers in Cardiology.

Poster Presentations

• Moukabary, T., & Haines, D. E. (2008, April). Effects of Acetylcholine Concentration on Action Potential Duration Restitution of Atrial Cells – A Simulation Study.. International Society for Computerized Electrocardiology (ISCE) Annual Conference. California.